Author

Martin Blohmer

Harvard UniversityORCID 0000-0002-5911-8420Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 2 Approach:statistical model· 2 Research Focus:variant effect prediction· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

David M. CheekiD· 2 shared Kamila NaxerovaiD· 2 shared Alexander N. GorelickiD· 1 shared Dejan JuriciD· 1 shared Djamila BoermaiD· 1 shared Emma C. E. WassenaariD· 1 shared Emma V. WatsoniD· 1 shared Emre KüçükköseiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCancer Biology

David M. CheekORCID

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2 papers

Recent·Most cited

Cancer BiologyNature GeneticsPeer-reviewedMay 5, 2026

Age distinguishes selection from causation in cancer genomes

David M. Cheek, Martin Blohmer, Martin A. Nowak…·Kamila Naxerova

The paper distinguishes between mutations that drive cancer and those that arise from normal tissue evolution by analyzing patient age across cancer and normal genomes.

5 citationstop 1%

humanwhole-genome sequencing

𝕏 PostBluesky

GeneticsbioRxivPreprintSep 25, 2024

A unique interplay of access and selection shapes peritoneal metastasis evolution in colorectal cancer

Emma C. E. Wassenaar, Alexander N. Gorelick, Wei-Ting Hung…·Kamila Naxerova

This study examines the evolutionary dynamics of peritoneal metastases in colorectal cancer, finding they are more heterogeneous than liver metastases but less diverse than locoregional metastases.

3 citations

humanstatistical model

𝕏 PostBluesky