Emma H. Wilson

This study identifies multiple bradyzoite subtypes in Toxoplasma cysts and shows they initiate distinct developmental pathways, revealing new insights into cyst formation and reactivation.

This paper shows that GCN5a loss in Toxoplasma gondii promotes latency by causing telomere dysfunction and altering gene expression during differentiation.

This study shows that Toxoplasma gondii infection of neurons reduces EV production and alters their content, leading to changes in astrocyte phenotype and GLT-1 loss.

The paper develops ex vivo bradyzoite-based methods to generate transgenic Toxoplasma gondii and identifies gene functions in bradyzoite reactivation pathways.