Author

Hannah E. Mercer

University of IowaORCID 0000-0002-6545-1545Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetics· 2 Molecular Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 4 Approach:statistical model· 4 Research Focus:cell type annotation· 2 Assay:imaging· 2 Research Focus:benchmarking· 1 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Kacie J. MeyeriD· 4 shared Michael G. AndersoniD· 4 shared Adam Hedberg‐BuenziD· 3 shared Carly J. van der HeideiD· 3 shared Danielle R. PellackiD· 2 shared Nicholas Pomernackas· 2 shared S. Scott WhitmoreiD· 2 shared Todd E. ScheetziD· 2 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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4 shared papersGenetics

Kacie J. MeyerORCID

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Author profile →

4 papers

Recent·Most cited

Genetic modifiers of Cep290-mediated retinal degeneration

Kacie J. Meyer, S. Scott Whitmore, Erin R. Burnight, Megan J. Riker, Hannah E. Mercer, A. Ajose, Gareth R. Howell, Edwin M. Stone, Robert F. Mullins, Budd A. Tucker, Todd E. Scheetz, Michael G. Anderson

The paper identifies genetic modifiers of CEP290-mediated retinal degeneration in mice using QTL analysis and genetic crosses.

bioRxiv · Jan 26, 2022 · PreprintGenetics

Tyr is Responsible for the Cctq1a QTL and Links Developmental Environment to Central Corneal Thickness Determination

Kacie J. Meyer, Demelza R. Larson, S. Scott Whitmore, Carly J. van der Heide, Adam Hedberg‐Buenz, Laura M. Dutca, Swanand Koli, Nicholas Pomernackas, Hannah E. Mercer, Maurisa Mansaray, William J. Paradee, Kai Wang, K. Saidas Nair, Todd E. Scheetz, Michael G. Anderson

The paper identifies Tyr as the gene responsible for the Cctq1a QTL and shows that developmental light exposure interacts with genotype to influence central corneal thickness.

bioRxiv · Sep 22, 2021 · PreprintGenetics

Quantification and image-derived phenotyping of retinal ganglion cell nuclei in the nee mouse model of congenital glaucoma

Carly J. van der Heide, Kacie J. Meyer, Adam Hedberg‐Buenz, Danielle R. Pellack, Nicholas Pomernackas, Hannah E. Mercer, Michael G. Anderson

This paper quantifies retinal ganglion cell loss and analyzes nuclear size changes in nee mice with congenital glaucoma using image-based phenotyping.

bioRxiv · Apr 13, 2021Molecular Biology

Biological Correlations and Confounding Variables for Quantification of Retinal Ganglion Cells Based on Optical Coherence Tomography Using Diversity Outbred Mice

Adam Hedberg‐Buenz, Kacie J. Meyer, Carly J. van der Heide, Wenxiang Deng, Kyungmoo Lee, Dana A. Soukup, Monica Kettelson, Danielle R. Pellack, Hannah E. Mercer, Kai Wang, Mona K. Garvin, Michael D. Abràmoff, Michael G. Anderson

This study compares OCT-derived GCC thickness to RGC abundance in Diversity Outbred mice, finding significant correlations between GCC thickness and RGC somal density and axon number.

bioRxiv · Dec 23, 2020 · PreprintNeuroscience