Author

Maja Matoga

ETH ZurichORCID 0000-0002-6622-4651Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Adriano AguzziiD· 1 shared Daniel JutziiD· 1 shared Florian MalardiD· 1 shared Frédéric H.‐T. AllainiD· 1 shared Jingjing GuoiD· 1 shared Ksenija RomaneiD· 1 shared Marc‐David RueppiD· 1 shared Marina KorotkovaiD· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperNeuroscience

Adriano AguzziORCID

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintApr 4, 2023

NG2 glia protect against prion neurotoxicity by inhibiting prostaglandin E2 signaling

Yingjun Liu, Jingjing Guo, Maja Matoga…·Adriano Aguzzi

This study shows NG2 glia reduce prion-induced neurodegeneration by inhibiting PGE2 signaling, which promotes microglial activation and neurotoxicity.

3 citations

mousebulk RNA-seq

Molecular BiologybioRxivPreprintAug 30, 2022

The cancer-associated RBM39 bridges the pre-mRNA, U1 and U2 snRNPs to regulate alternative splicing

Sébastien Campagne, Daniel Jutzi, Florian Malard…·Frédéric H.‐T. Allain

This study identifies RBM39's role in autoregulating its own pre-mRNA splicing through interactions with U1 and U2 snRNPs, offering insights for alternative cancer therapies.

DOI 10.1101/2022.08.30.505862

humanbulk RNA-seq