Author

Koh‐ichi Nagata

Aichi Developmental Disability CenterORCID 0000-0002-6827-8434Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 1 Organism:mouse· 1

Frequent Co-Authors

Ben TitleiD· 1 shared Bjorg GudmundsdottiriD· 1 shared Galya Monderer‐Rothkoff· 1 shared Guo‐Jen HuangiD· 1 shared Jonathan R. KelleriD· 1 shared Kristbjorn O. GudmundssoniD· 1 shared Maayan Tal· 1 shared Nanako HamadaiD· 1 shared

Activity by Year

2020-2020 · peak 1/yr

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10 top collaborators · 45 mutual co-author links

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1 shared paperGenetics

Ben TitleORCID

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1 paper

Recent·Most cited

GeneticsNature CommunicationsPeer-reviewedNov 17, 2020

Pogz deficiency leads to abnormal behavior, transcription dysregulation and impaired cerebellar physiology

Reut Suliman-Lavie, Ben Title, Yahel Cohen…·Sagiv Shifman

This study shows that Pogz deficiency in mice causes brain abnormalities, transcription dysregulation, and cerebellar dysfunction resembling ASD features in humans.

58 citationstop 3%

mouseexperimental assay