Author

Bjorg Gudmundsdottir

National Institutes of HealthORCID 0000-0002-1236-3410Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Kristbjorn O. GudmundssoniD· 2 shared Ben TitleiD· 1 shared Galya Monderer‐Rothkoff· 1 shared Guo‐Jen HuangiD· 1 shared John F. TisdaleiD· 1 shared Jonathan R. KelleriD· 1 shared Kartik R. RoyiD· 1 shared Koh‐ichi NagataiD· 1 shared

Activity by Year

2020-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersDevelopmental Biology

Kristbjorn O. GudmundssonORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Developmental BiologybioRxivPreprintMar 24, 2025

MLL1 complex is a critical regulator of fetal hemoglobin repression

Yufen Han, Bjorg Gudmundsdottir, Kristbjorn O. Gudmundsson…·Yang Du

This study identifies the MLL1 complex as a regulator of fetal hemoglobin repression and shows that targeting MEN1 or KMT2A increases fetal hemoglobin expression in erythroid cells.

DOI 10.1101/2025.03.24.645036

humanbulk RNA-seq

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GeneticsNature CommunicationsPeer-reviewedNov 17, 2020

Pogz deficiency leads to abnormal behavior, transcription dysregulation and impaired cerebellar physiology

Reut Suliman-Lavie, Ben Title, Yahel Cohen…·Sagiv Shifman

This study shows that Pogz deficiency in mice causes brain abnormalities, transcription dysregulation, and cerebellar dysfunction resembling ASD features in humans.

58 citationstop 3%

mouseexperimental assay

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