Author

Hidekazu Sotoyama

Brain Research Institute, Niigata UniversityORCID 0000-0002-7206-0454Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Assay:electronic health records· 1

Frequent Co-Authors

Hiroyoshi InabaiD· 1 shared Hiroyuki NawaiD· 1 shared Hisaaki NambaiD· 1 shared Kazuya IwamotoiD· 1 shared Y. KobayashiiD· 1 shared Yui MurataiD· 1 shared Yuriko IwakuraiD· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

7 top collaborators · 21 mutual co-author links

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1 shared paperNeuroscience

Hiroyoshi InabaORCID

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintMay 9, 2023

Neural mechanisms underlying release-mode-specific abnormalities in dopamine neural activity in a schizophrenia-like model

Hidekazu Sotoyama

This paper investigates dopamine release mode abnormalities in a schizophrenia-like model and identifies mechanisms involving serotonergic regulation and antipsychotic effects.

DOI 10.1101/2023.05.09.540082

mouseelectronic health records

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NeuroscienceNeuropsychopharmacology ReportsPeer-reviewedMay 27, 2021

Inter-breeder differences in prepulse inhibition deficits of C57BL/6J mice in a maternal infection model for schizophrenia

Y. Kobayashi, Hiroyoshi Inaba, Yuriko Iwakura…·Hiroyuki Nawa

This study shows that maternal infection with Poly(I:C) affects prepulse inhibition in offspring differently depending on the breeder, but not startle response or adaptation.

15 citations

mouseexperimental assay

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