Author

Mitchell P. Goldfarb

The Graduate Center, CUNYORCID 0000-0002-7219-7645Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Assay:electronic health records· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Assay:electrophysiology· 1

Frequent Co-Authors

Akshay SharmaiD· 1 shared Christopher MarraiD· 1 shared Christopher MarraiD· 1 shared Egidio Ugo D'AngeloiD· 1 shared Francesca LocatelliiD· 1 shared Francesca PrestoriiD· 1 shared Laura BottaiD· 1 shared Lillian LawrenceiD· 1 shared

Activity by Year

2019-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperPhysiology

Akshay SharmaORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Carboxyl Terminal Domain Missense Mutations Alter Distinct Gating Properties of the Cardiac Sodium Channel

Akshay Sharma, Christopher Marra, Nomon Mohammad, Vasilisa Iatckova, Lillian Lawrence, Mitchell P. Goldfarb

This study investigates how missense mutations in the CTD of Nav1.5 affect gating properties, revealing distinct effects on inactivation and activation.

The Journal of General Physiology · Oct 23, 2025Peer-reviewedPhysiology

Enhanced Sodium Channel Inactivation by Temperature and FHF2 Deficiency Blocks Heat Nociception

Christopher Marra, Timothy V. Hartke, Matthias Ringkamp, Mitchell P. Goldfarb

The paper shows that FHF2 deficiency and elevated temperature together block heat nociception by altering sodium channel inactivation in dorsal root ganglion C fibers.

Pain · Nov 9, 2022Peer-reviewedNeuroscience

Hyper-excitability and hyper-plasticity disrupt cerebellar signal transfer in the IB2 KO mouse model of autism

Egidio Ugo D'Angelo, Teresa Soda, Lisa Mapelli, Francesca Locatelli, Laura Botta, Mitchell P. Goldfarb, Francesca Prestori

The paper investigates neuronal and circuit properties in IB2 KO mice, showing hyper-excitability and hyper-plasticity disrupt cerebellar signal transfer, supporting cerebellar involvement in ASD.

Journal of Neuroscience · Jan 29, 2019Peer-reviewedNeuroscience