Author

Yuji Kamioka

Kansai Medical UniversityORCID 0000-0002-7532-1787Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Organism:human· 1

Frequent Co-Authors

Ami Fukushi-Kumagaya· 1 shared Asako Katsuma· 1 shared Daisuke KawauchiiD· 1 shared Hiroko Katsuno‐KambeiD· 1 shared Kentarou BabaiD· 1 shared Megumi Morisaki· 1 shared Mizuki Sakai· 1 shared Naoyuki InagakiiD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCell Biology

Ami Fukushi-Kumagaya

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1 paper

Recent·Most cited

Weak and tunable adhesion-clutch drives rapid cell migration and glioblastoma motility

Kentarou Baba, Ami Fukushi-Kumagaya, Megumi Morisaki, Ryosuke F. Takeuchi, Zhijie Xiao, Yoshikazu Nagashima, Mizuki Sakai, Yasuna Higashiguchi, Hiroko Katsuno‐Kambe, Asako Katsuma, Yoshihiro Ueda, Yuji Kamioka, Daisuke Kawauchi, Tatsuo Kinashi, Yonehiro Kanemura, Naoyuki Inagaki

The paper identifies a weak, integrin-independent adhesion-clutch mechanism involving shootin1 and L1 that enables rapid cell migration and glioblastoma motility by transmitting weak traction forces.

bioRxiv · Jan 13, 2025 · PreprintCell Biology