Author

José Manuel Cóndor Capcha

University of MiamiORCID 0000-0002-8005-6244Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:human· 1 Research Focus:metabolism & bioenergetics· 1 Organism:rat· 1

Frequent Co-Authors

Lina A. ShehadehiD· 2 shared Ahmed ChahdiiD· 1 shared Aisha KhaniD· 1 shared Ali KamiariD· 1 shared Anastasios LymperopoulosiD· 1 shared Camila Iansen IrioniD· 1 shared Carolina RiveraiD· 1 shared Chryso Pefkaros KatsoufisiD· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCell Biology

Lina A. ShehadehORCID

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2 papers

Recent·Most cited

Cell BiologybioRxivPreprintFeb 20, 2024

Sarcomeric SRX:DRX Equilibrium in Alport and LDLR/P407 Mouse Models of HFpEF.

Ali Kamiar, Monique M. Williams, José Manuel Cóndor Capcha…·Lina A. Shehadeh

The paper quantifies SRX and DRX myosin states in HFpEF mouse models, finding altered SRX:DRX proportions linked to specific cardiac phenotypes.

DOI 10.1101/2024.02.20.581314

mouseexperimental assay

𝕏 PostBluesky

Molecular BiologybioRxivPreprintNov 6, 2020

βPix sequesters IDOL and prevents LDL receptor degradation through a β2AR-regulated signaling pathway in Alport Syndrome

Ahmed Chahdi, Keyvan Yousefi, José Manuel Cóndor Capcha…·Lina A. Shehadeh

This study identifies βPix as a regulator of LDLR stability by sequestering IDOL, preventing LDLR degradation, and reducing LDL-C uptake in Alport Syndrome models.

DOI 10.1101/2020.11.06.372292

𝕏 PostBluesky