Author

Koichi Utani

Kanazawa Medical UniversityORCID 0000-0002-8065-3161Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Genomics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:chromatin & epigenetic regulation· 1 Organism:human· 1

Frequent Co-Authors

Adrian M. BarisiD· 1 shared Anna B. Marks· 1 shared Bhushan L. ThakuriD· 1 shared Christophe E. RedoniD· 1 shared Haiqing FuiD· 1 shared Jacob GrossiD· 1 shared Kuniyoshi IwabuchiiD· 1 shared Lőrinc Sándor PongoriD· 1 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperGenomics

Adrian M. BarisORCID

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2 papers

Recent·Most cited

USP10 Facilitates Homologous Recombination-Mediated DNA Double-Strand Break Repair through Localization to the Nucleolus

Koichi Utani, Ryo Sakasai, Toshiki Himeda, Takako Okuwa, Kuniyoshi Iwabuchi, Masaya Higuchi

This study shows USP10 is needed for homologous recombination repair of DNA breaks and genomic stability by localizing to the nucleolus.

bioRxiv · May 11, 2026 · PreprintCell Biology

Dynamics of replication origin over-activation

Haiqing Fu, Christophe E. Redon, Bhushan L. Thakur, Koichi Utani, Robin Sebastian, Sang‐Min Jang, Jacob Gross, Sara Mosavarpour, Anna B. Marks, Sophie Z. Zhuang, Sarah B. Lazar, Mishal Rao, Shira T. Mencer, Adrian M. Baris, Lőrinc Sándor Pongor, Mirit I. Aladjem

The paper investigates replication origin over-activation in cancer cells and finds two distinct pools of origins are activated under different conditions.

Nature Communications · Jun 8, 2021Peer-reviewedGenomics