Author

Samuel Gritz

Rutgers, The State University of New JerseyORCID 0000-0002-8071-119XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Assay:electronic health records· 2 Organism:mouse· 2 Research Focus:benchmarking· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Research Focus:spatial domain detection· 1 Approach:statistical model· 1

Frequent Co-Authors

Aaron D. MilsteiniD· 2 shared Alessandro R. GalloniiD· 1 shared Anshul Voleti· 1 shared Matthew S. ScarnatiiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

4 top collaborators · 6 mutual co-author links

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2 shared papersNeuroscience

Aaron D. MilsteinORCID

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2 papers

Recent·Most cited

GIRK Channel Loss of Function Increases Dendritic Excitability in a Mouse Model of GNB1 Encephalopathy

Samuel Gritz, Anshul Voleti, Matthew S. Scarnati, Alessandro R. Galloni, Aaron D. Milstein

This study shows that a GNB1 mutation impairs GIRK channel function in mice, leading to increased dendritic excitability and seizure susceptibility, which can be reversed by a GIRK activator.

bioRxiv · Aug 14, 2026 · PreprintNeuroscience

Hippocampal Place Cells with NMDARs Do Not Require Excitation and Inhibition to Be Reciprocally Tuned

Samuel Gritz, Aaron D. Milstein

The paper shows that NMDARs allow place cells to maintain spatial selectivity without requiring changes in inhibitory inputs.

bioRxiv · Apr 29, 2026 · PreprintNeuroscience