Author

Richard D Iggo

Université de BordeauxORCID 0000-0002-8822-9111Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1

Frequent Co-Authors

Alexandre FavereauxiD· 1 shared Amy R. DwyeriD· 1 shared Anne‐Aurélie RaymondiD· 1 shared Anthony BergeroniD· 1 shared Anthony BouteriD· 1 shared Cyril DourtheiD· 1 shared Ebtihal H MustafaiD· 1 shared Élodie RichardiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCell Biology

Alexandre FavereauxORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

The transcription factor AP-2β defines active enhancers conferring molecular apocrine cell identity in breast cancer

Ebtihal H Mustafa, Geraldine Laven‐Law, Jean M. Winter, Zoya Kikhtyak, Anthony Bergeron, Gaëtan Marie MacGrogan, Amy R. Dwyer, Stephen Pederson, Wayne D. Tilley, Richard D Iggo, Theresa E. Hickey

This study identifies AP-2β as a key regulator of active enhancers in molecular apocrine breast cancer, linking its expression to cell identity and proliferation.

bioRxiv · Jul 5, 2025 · PreprintCancer Biology

Cancer cells transfer invasive properties through microRNAs contained in collagen-tracks

Léa Normand, Lucile Rouyer, Élodie Richard, Nathalie Allain, Julie Giraud, Sylvaine Di-Tommaso, Cyril Dourthe, Anne‐Aurélie Raymond, Jean‐William Dupuy, Kévin Moreau, Richard D Iggo, Gaetan McGrogan, Nathalie Dugot‐Senant, Anthony Bouter, Sisareuth Tan, , Frédéric Saltel

The paper identifies collagen-tracks as a new form of cell-cell communication that transfer invasive properties via miRNAs, promoting metastasis in breast cancer.

bioRxiv · Jan 25, 2025 · PreprintCell Biology