Timothy S. Balmer

This study investigates the role of NMDA receptor signaling in the development and function of cerebellar unipolar brush cells, finding that NMDARs are not essential for dendritic development but may regulate cell death and excitability.

The paper investigates how group II mGluRs modulate the sodium leak channel NALCN in cerebellar unipolar brush cells, revealing a novel mechanism of glutamatergic synaptic inhibition.

This study shows that unipolar brush cells (UBCs) are essential for balance in aged mice, as disrupting their activity impairs balance performance and vestibular-related behaviors in older mice but not young mice.

This study compares six AAV serotypes in mouse brain regions, finding AAV1 provides the highest transgene expression and labels more cell types than other serotypes.

The paper investigates feed-forward cerebellar circuits involving unipolar brush cells (UBCs) and shows they extend and diversify sensory signaling through mutual innervation and computational modeling.

The paper shows that ambient glutamate in the synaptic cleft modulates synaptic responses in cerebellar unipolar brush cells through glutamate transporters.

The paper shows that primary afferents selectively connect to ON unipolar brush cells, while secondary afferents connect to both ON and OFF subtypes, revealing distinct anatomical pathways for signal transformation.

The paper shows that TrkB activation during a critical period maintains receptive field refinement in adult superior colliculus, mimicking the protective effects of early visual experience.