Author

Maria Almira Correia

University of California, San FranciscoORCID 0000-0002-8939-2180Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Organism:mouse· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

Liang HeiD· 2 shared Alma L. BurlingameiD· 1 shared Doyoung KwoniD· 1 shared Eric F. JohnsoniD· 1 shared Hong XiaoiD· 1 shared Jade YangiD· 1 shared Jinyu LuoiD· 1 shared Kathy H. LiiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCell Biology

Liang HeORCID

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2 papers

Recent·Most cited

Hepatic cytochrome P450 endoplasmic reticulum-associated degradation (ERAD): Topological determinants and cellular partnerships that dictate the preferential P450 proteolytic sorting into macroautophagy rather than UPS.

Hong Xiao, Liang He, Yi Liu, Jinyu Luo, Xiaokun Shu, Maria Almira Correia

This study identifies how the topology of ER-microdomains and interactions with erlin-1 determine the proteolytic fate of P450s via ERAD or ERLAD.

bioRxiv · Sep 25, 2025 · PreprintCell Biology

Liver CYP4A autophagic-lysosomal degradation (ALD): A major role for the autophagic receptor SQSTM1/p62 through an uncommon target interaction site

Liang He, Doyoung Kwon, Michael J. Trnka, Yi Liu, Jade Yang, Kathy H. Li, Rheem A. Totah, Eric F. Johnson, Alma L. Burlingame, Maria Almira Correia

The paper identifies SQSTM1/p62 as a key autophagic receptor for CYP4A degradation, showing its role in preventing proinflammatory metabolite production and insoluble aggregates in liver cells.

Molecular Pharmacology · Jul 15, 2025Peer-reviewedCell Biology