Author

Mingyu Lee

Seoul National UniversityORCID 0000-0002-8970-1895Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:benchmarking· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:machine learning (classical)· 1 Assay:multiome· 1

Frequent Co-Authors

Aakansha RaiiD· 1 shared Ádám Misák· 1 shared Ah Reum KimiD· 1 shared Aida Nourbakhsh· 1 shared Akash Gosain· 1 shared Alexander Wu· 1 shared Alice DriesseniD· 1 shared Alidivinas Prusokas· 1 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperBioinformatics

Aakansha RaiORCID

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2 papers

Recent·Most cited

Cell-to-cell and type-to-type heterogeneity of signaling networks: Insights from the crowd

Attila Gábor, Marco Tognetti, Alice Driessen, Jovan Tanevski, Baosen Guo, Wencai Cao, He Shen, Thomas Von Yu, Verena Chung, Bernd Bodenmiller, Julio Sáez-Rodríguez, Augustinas Prusokas, Alidivinas Prusokas, Renata Retkutė, Anand Rajasekar, , Mingyu Lee

The paper organizes a DREAM challenge to benchmark predictive models of single-cell signaling using mass cytometry data from breast cancer cell lines.

Molecular Systems Biology · Oct 1, 2021Peer-reviewedBioinformatics

Mutations in TMEM43 cause autosomal dominant auditory neuropathy spectrum disorder via interaction with Connexin-mediated passive conductance channels

Minwoo Wendy Jang, Doo‐Yi Oh, Eunyoung Yi, Xuezhong Liu, Jie Ling, Nayoung Kim, Kushal Sharma, Tai Young Kim, Seungmin Lee, Ah Reum Kim, Min Young Kim, Min A Kim, Mingyu Lee, Jin Hee Han, Jae Joon Han, , Byung Yoon Choi

This study identifies TMEM43 mutations as a cause of autosomal dominant auditory neuropathy spectrum disorder by showing their interaction with Connexin-mediated channels in cochlear glia-like supporting cells.

Proceedings of the National Academy of Sciences · May 28, 2021Peer-reviewedGenetics