Author

Sofia Stenler

University of OxfordORCID 0000-0002-9017-3030Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:protein structure prediction· 1

Frequent Co-Authors

Anna M. L. Coenen-StassiD· 2 shared Matthew J. A. WoodiD· 2 shared Thomas C. RobertsiD· 2 shared Britt HansoniD· 1 shared Junyu Huang· 1 shared Kaarel KrjutškoviD· 1 shared Katarzyna Chwalenia· 1 shared Marc S. WeinbergiD· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersNeuroscience

Anna M. L. Coenen-StassORCID

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2 papers

Recent·Most cited

Timing matters: exon skipping therapy is most effective when initiated early in a mouse model of Duchenne muscular dystrophy

Sofia Stenler, Junyu Huang, Tirsa L.E. van Westering, Anna M. L. Coenen-Stass, Yahya E. Jad, Kaarel Krjutškov, Samir EL Andaloussi, Matthew J. A. Wood, Thomas C. Roberts

This study shows that exon skipping therapy is more effective when started early in adult mdx mice than in aged mice, leading to better dystrophin restoration and transcriptomic changes.

Molecular Therapy — Nucleic Acids · Jul 17, 2026Peer-reviewedNeuroscience

Non-uniform dystrophin re-expression after CRISPR-mediated exon excision in the dystrophin/utrophin double-knockout mouse model of DMD

Britt Hanson, Sofia Stenler, Nina Ahlskog, Katarzyna Chwalenia, Nenad Svrzikapa, Anna M. L. Coenen-Stass, Marc S. Weinberg, Matthew J. A. Wood, Thomas C. Roberts

This study uses CRISPR to excise exon 23 in dKO mice, achieving partial dystrophin restoration but with limited efficacy and challenges in DNA repair.

Molecular Therapy — Nucleic Acids · Oct 23, 2022Peer-reviewedMolecular Biology