Author

Yasuyoshi Tanaka

Daiichi University of PharmacyORCID 0000-0002-9053-4342Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Assay:electronic health records· 1

Frequent Co-Authors

Kaori KubotaiD· 2 shared Katsunori Iwasaki· 2 shared Kouya UchinoiD· 2 shared Masanobu Deshimaru· 2 shared Shinichi HiroseiD· 2 shared Shutaro KatsurabayashiiD· 2 shared Takuya WatanabeiD· 2 shared Ai Asahara· 1 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 43 mutual co-author links

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2 shared papersNeuroscience

Kaori KubotaORCID

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2 papers

Recent·Most cited

Inhibitory synaptic transmission is impaired at higher extracellular Ca2+ concentrations in Scn1a+/- mouse model of Dravet syndrome

Kouya Uchino, Hiroyuki Kawano, Yasuyoshi Tanaka, Yuna Adaniya, Ai Asahara, Masanobu Deshimaru, Kaori Kubota, Takuya Watanabe, Shutaro Katsurabayashi, Katsunori Iwasaki, Shinichi Hirose

This study shows that inhibitory synaptic transmission is impaired in Scn1a+/- mice, with reduced inhibitory synapses and increased sensitivity to extracellular Ca2+.

Scientific Reports · May 20, 2021Peer-reviewedNeuroscience

Astrocyte Ca2+ Signaling is Facilitated in an Scn1a+/- Mouse Model of Dravet Syndrome

Kouya Uchino, Yasuyoshi Tanaka, Wakana Ikezawa, Masanobu Deshimaru, Kaori Kubota, Takuya Watanabe, Shutaro Katsurabayashi, Katsunori Iwasaki, Shinichi Hirose

This study shows that astrocyte Ca2+ signaling is enhanced in Scn1a+/- mice, suggesting a role for astrocytic Ca2+ dysregulation in Dravet syndrome pathogenesis.

bioRxiv · May 18, 2021Neuroscience