Author

Kouya Uchino

Fukuoka UniversityORCID 0000-0003-1300-8469Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Assay:electronic health records· 1

Frequent Co-Authors

Kaori KubotaiD· 2 shared Katsunori Iwasaki· 2 shared Masanobu Deshimaru· 2 shared Shinichi HiroseiD· 2 shared Shutaro KatsurabayashiiD· 2 shared Takuya WatanabeiD· 2 shared Yasuyoshi TanakaiD· 2 shared Ai Asahara· 1 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 43 mutual co-author links

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2 shared papersNeuroscience

Kaori KubotaORCID

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2 papers

Recent·Most cited

NeuroscienceScientific ReportsPeer-reviewedMay 20, 2021

Inhibitory synaptic transmission is impaired at higher extracellular Ca2+ concentrations in Scn1a+/- mouse model of Dravet syndrome

Kouya Uchino, Hiroyuki Kawano, Yasuyoshi Tanaka…·Shinichi Hirose

This study shows that inhibitory synaptic transmission is impaired in Scn1a+/- mice, with reduced inhibitory synapses and increased sensitivity to extracellular Ca2+.

15 citations

mouseelectronic health records

𝕏 PostBluesky

NeurosciencebioRxivMay 18, 2021

Astrocyte Ca2+ Signaling is Facilitated in an Scn1a+/- Mouse Model of Dravet Syndrome

Kouya Uchino, Yasuyoshi Tanaka, Wakana Ikezawa…·Shinichi Hirose

This study shows that astrocyte Ca2+ signaling is enhanced in Scn1a+/- mice, suggesting a role for astrocytic Ca2+ dysregulation in Dravet syndrome pathogenesis.

10 citations

mouseexperimental assay

𝕏 PostBluesky