Wu‐Min Deng

The paper uses live imaging to study epithelial cell transformation into phagocytes in Drosophila ovaries, revealing new transient behaviors in phagocytic processes.

This study identifies leptin as a peripheral signal that modulates steroidogenesis by altering lipid pools through JAK/STAT signaling across species.

This study shows tumors induce species-specific microbiome changes that trigger renal inflammation and ascites in Drosophila.

This study shows that polyploidy, induced by Notch signaling, enables epithelial cells to become non-professional phagocytes during stress, facilitating phagocytic functions essential for tissue homeostasis.

This study identifies Topoisomerase I as a conserved regulator of satellite DNA transcription across species, showing its role in centromere function and DNA damage response.

This study identifies FruCOM and Hnf4 as regulators of pheromone biosynthesis and perception in Drosophila, linking lipid metabolism to sexual attraction.

The paper uses single-cell RNA sequencing to study polarity loss in Drosophila follicle cells and identifies Keap1-Nrf2 signaling as promoting invasive behavior.

This study uses single-cell RNA sequencing to analyze the adult Drosophila ovary, identifying transcriptional programs that govern oogenesis and revealing new genetic markers and transcriptional signatures.

The paper identifies a developmentally regulated process called patency in Drosophila follicular epithelium that facilitates lipid uptake and is exploited by endosymbionts.

This study shows that the piRNA pathway suppresses UASt expression in Drosophila germline cells by targeting the hsp70 promoter, while the SV40 3UTR is not involved in this suppression.