Author

Camille L. Novis

University of UtahORCID 0000-0002-9111-3662Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Organism:mouse· 1

Frequent Co-Authors

Andrew BaessleriD· 1 shared Andrew W. RichensiD· 1 shared Brittany Winter· 1 shared Bryant PerkinsiD· 1 shared Bryce Fuchs· 1 shared J. Scott HaleiD· 1 shared Kaitlyn A. FlintiD· 1 shared Katherine Elena VarleyiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperImmunology

Andrew BaesslerORCID

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1 paper

Recent·Most cited

Dnmt3a-mediated de novo DNA methylation enforces lineage commitment and preserves functionality of memory Th1 and Tfh cells

Bryant Perkins, Camille L. Novis, Andrew Baessler, Linda M. Sircy, M. Thomas, Malia H. Harrison-Chau, Andrew W. Richens, Bryce Fuchs, Nguyen X. Nguyen, Kaitlyn A. Flint, Brittany Winter, Katherine Elena Varley, J. Scott Hale

This paper shows that Dnmt3a-mediated DNA methylation is essential for maintaining lineage commitment and function of memory Th1 and Tfh cells, and that early inhibition of this process can enhance Tfh cell differentiation.

Cell Reports · Mar 1, 2026Peer-reviewedImmunology