Camille E. A. Goo

This study shows that loss of polr1c in zebrafish causes reduced myelination and craniofacial defects, with Tp53 pathway activation but no apoptosis in oligodendrocyte precursors.

The paper identifies dendritic atoh1a+ cells as transient intermediates in zebrafish Merkel cell development and regeneration, showing their role in morphology acquisition through actin remodeling and Ectodysplasin A signaling.

This study identifies Merkel cells in zebrafish, shows their origin and characteristics, and demonstrates their relationship with dermal appendages during skin development.