Darukeshwara Joladarashi

This study identifies that Podoplanin-expressing mesenchymal stromal cells release exosomes that trigger SAA3 overproduction in macrophages, leading to cardiac amyloidosis after myocardial infarction.

This study shows circ-Cdr1as modulates macrophage phenotype and cardiac repair via the circ-Cdr1as-miR-7-Klf4 pathway, improving cardiac function post-myocardial infarction.