Author

Peter van der Meer

University Medical Center GroningenORCID 0000-0002-9705-4413Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Immunology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Adriaan A. VoorsiD· 1 shared Alejandra Isasi Nalvarte· 1 shared Anna M. FeringaiD· 1 shared Derk FrankiD· 1 shared Elisabeth M. SchouteniD· 1 shared Ferdy LambregtseiD· 1 shared Florian LeuschneriD· 1 shared Florian SicklingeriD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperImmunology

Adriaan A. VoorsORCID

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2 papers

Recent·Most cited

Dietary selenium deficiency drives sex-specific circadian disturbance through redox imbalance and causes early systolic dysfunction in mice

Xuanxuan Guo, Ferdy Lambregtse, Lotte Geerlings, Jumo Zhu, Sietske N. Zijlstra, Anna M. Feringa, Elisabeth M. Schouten, Herman H.W. Silljé, Lutz Schomburg, Peter van der Meer, Nils Bömer

This study shows that selenium deficiency in mice causes early systolic dysfunction and molecular changes linked to redox imbalance and circadian disruption, with sex-specific effects.

bioRxiv · Sep 29, 2026 · PreprintMolecular Biology

Tricuspid valve regurgitation accelerates heart failure via a cardio-intestinal innate immune circuit

Florian Sicklinger, Tobias Thiemann, Sarah Rupprecht, Lisa Quadt, Junedh Amrute, Jan Zuchgan, Jakob Christoph Voran, George Markousis‐Mavrogenis, Alejandra Isasi Nalvarte, Laura M. Wienecke, Niklas Hartmann, S Erbe, Isabel A. Hoerbrand, Martin Kraus, Martin J. Gruber, , Florian Leuschner

This study identifies a cardio-intestinal innate immune circuit driven by tricuspid valve regurgitation that promotes heart failure through IL-6-mediated myocardial fibrosis and dysfunction.

bioRxiv · Jul 11, 2026 · PreprintImmunology