Author

Jonathan Chuck

Scripps Research InstituteORCID 0000-0003-0081-4330Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1 Research Focus:protein folding & dynamics· 1 Assay:proteomics· 1

Frequent Co-Authors

Christopher C. Williams· 1 shared Douglas J. KojetiniD· 1 shared Laura A. SoltiD· 1 shared Mohammed AmiriD· 1 shared Paola Munoz‐TelloiD· 1 shared Qun LuiD· 1 shared Sarah A. MosureiD· 1 shared Sean Campbell· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

8 top collaborators · 13 mutual co-author links

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1 shared paperBiophysics

Christopher C. Williams

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2 papers

Recent·Most cited

ImmunologybioRxivPreprintJan 28, 2026

Negative regulation of TH17-mediated inflammation by the nuclear receptor REV-ERBβ

Sean Campbell, Sarah A. Mosure, Mohammed Amir…·Laura A. Solt

This study shows REV-ERBβ negatively regulates TH17-mediated inflammation in mouse models, acting independently from REV-ERB through distinct mechanisms.

DOI 10.64898/2026.01.26.701826

mousebulk RNA-seq

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BiophysicsProceedings of the National Academy of SciencesPeer-reviewedMay 19, 2025

A tethering mechanism underlies Pin1-catalyzed proline cis-trans isomerization at a noncanonical site

Christopher C. Williams, Jonathan Chuck, Paola Munoz‐Tello, Douglas J. Kojetin

This paper identifies a tethering mechanism by which Pin1 catalyzes proline cis-trans isomerization at a noncanonical site in the PPARγ AF-1 domain.

1 citation

humanproteomics

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