Author

Francesca Fasanella Masci

University of GlasgowORCID 0000-0003-0120-7225Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Assay:proteomics· 1

Frequent Co-Authors

Elżbieta KaniaiD· 2 shared Evie RigbyiD· 2 shared Karen KeeshaniD· 2 shared Lijun Liu· 2 shared Akshara Narayanan· 1 shared Alastair SmithiD· 1 shared Alexander Winton· 1 shared Alexandra Curran· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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2 shared papersCancer Biology

Elżbieta KaniaORCID

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintApr 27, 2026

CD180 identifies chemoresistant stem-like blasts and reveals a KMT2A-driven vulnerability in acute myeloid leukaemia

Madonna M. Eltoukhy, Alexander Winton, Francesca Fasanella Masci…·Karen Keeshan

This paper identifies CD180 as a marker for chemoresistant stem-like blasts in KMT2A-rearranged paediatric AML, showing its absence on normal HSCs and its role as a therapeutic target.

DOI 10.64898/2026.04.23.720316

humanscRNA-seq

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Cancer BiologyBiochemical JournalPeer-reviewedMar 13, 2026

Inducing TRIB2 Targeted Protein Degradation to Reverse Chemoresistance in Acute Myeloid Leukemia

Evie Rigby, Francesca Fasanella Masci, Akshara Narayanan…·Karen Keeshan

This study identifies TRIB2 as a therapeutic target in AML by demonstrating that its degradation via PROTACs and small molecules can reverse chemoresistance and enhance chemotherapy efficacy.

2 citations

humanproteomics

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