Author

Lijun Liu

University of Glasgow

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

Biology Disciplines

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Cancer Biology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:deep learning· 1

Frequent Co-Authors

Elżbieta KaniaiD· 2 shared Evie RigbyiD· 2 shared Francesca Fasanella MasciiD· 2 shared Karen KeeshaniD· 2 shared Ahmet Catak· 1 shared Aishwarya Ponna· 1 shared Akshara Narayanan· 1 shared Alastair SmithiD· 1 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 26 mutual co-author links

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2 shared papersCancer Biology

Elżbieta KaniaORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

The SET1/COMPASS subunit RBBP5 orchestrates epigenetic control of global proteostasis and the 12h oscillator to safeguard metabolic and cellular homeostasis

Syeda Kubra, Michelle Sun, William A. Dion, Yu Bian, Ahmet Catak, Hannah Luong, Haokun Wang, Yinghong Pan, Jia-Jun Liu, Aishwarya Ponna, Lijun Liu, IAN J. SIPULA, Jianhua Luo, Michael J. Jurczak, Silvia Liu, Bokai Zhu

This study identifies RBBP5 as an essential epigenetic regulator of the 12h oscillator and proteostasis, linking it to metabolic and cellular homeostasis in mammals.

PLoS Biology · Oct 8, 2026Peer-reviewedCell Biology

CD180 identifies chemoresistant stem-like blasts and reveals a KMT2A-driven vulnerability in acute myeloid leukaemia

Madonna M. Eltoukhy, Alexander Winton, Francesca Fasanella Masci, Elżbieta Kania, Mary T. Scott, Alastair Smith, Evie Rigby, Alexandra Curran, Aya Gouma, Jennifer E. Cassels, Lijun Liu, Thomas Stevens, Karen M. Dunn, Kevin M. Rattigan, Meaad almowaled, , Karen Keeshan

This paper identifies CD180 as a marker for chemoresistant stem-like blasts in KMT2A-rearranged paediatric AML, showing its absence on normal HSCs and its role as a therapeutic target.

bioRxiv · Apr 27, 2026 · PreprintCancer Biology

Inducing TRIB2 Targeted Protein Degradation to Reverse Chemoresistance in Acute Myeloid Leukemia

Evie Rigby, Francesca Fasanella Masci, Akshara Narayanan, Elżbieta Kania, John A. Harris, Jamie J.L. Williams, Binghua Zhang, Lijun Liu, Laura Richmond, Fengtao Zhou, Ke Ding, Ruaidhrı́ J. Carmody, Patrick A. Eyers, Karen Keeshan

This study identifies TRIB2 as a therapeutic target in AML by demonstrating that its degradation via PROTACs and small molecules can reverse chemoresistance and enhance chemotherapy efficacy.

Biochemical Journal · Mar 13, 2026Peer-reviewedCancer Biology