Dean G. Campelj

The paper identifies ADSS1 as a suppressed metabolic control node in dystrophic muscle, showing its role in purine metabolism and its impact on inflammatory and adipogenic remodelling.

This study shows that exercise training restores mitochondrial quality control in sarcopenic muscle by improving mitophagy, mitochondrial respiration, and muscle strength.

This study shows that BAM15 improves mitochondrial efficiency and contractile function in aged male mice but not females, suggesting sex-specific effects in sarcopenia.

This study shows that the AML chemotherapy regimen induces cachexia in mice, characterized by body and lean mass loss, and identifies haptoglobin as a potential biomarker for chemotherapy-induced cachexia.