Jennifer Loros

The paper identifies cell-autonomous and systemic circadian gene expression programs in adipocytes using RNA sequencing and motif analysis, revealing distinct regulatory programs linked to metabolic processes.

This study investigates the role of phosphatase PP4 in the Neurospora circadian clock, showing it regulates period length and compensation mechanisms.

The paper identifies protein phosphatases involved in circadian period, temperature compensation, and output in Neurospora by analyzing 39 non-essential phosphatases.

This study shows that rhythmic nuclear import mediated by importins regulates the Neurospora circadian clock by affecting FRQ and WCC dynamics and timing.

The paper investigates the role of kinases in temperature compensation of circadian rhythms in Neurospora, finding that Casein Kinase I and II have temperature-dependent regulatory roles on FRQ phosphorylation.

The paper tracks circadian clock protein dynamics in syncytia, revealing coordinated nuclear localization and subnuclear interactions across multiple nuclei.

The paper shows that WCC dephosphorylation in Neurospora sustains circadian rhythms without new WCC synthesis, highlighting phosphorylation as key to the circadian oscillator.

This study identifies novel regulators of Nutritional Compensation in the circadian clock of Neurospora crassa and shows conservation in mammalian cells.

This study identifies NCA-2 as a regulator of circadian period length in Neurospora by modulating intracellular calcium levels and affecting core clock component phosphorylation.

This study identifies Period-2 and Nonsense Mediated Decay as regulators of CKI levels, affecting circadian period in Neurospora crassa.

The paper introduces PIRS, a method to identify invariant genes from time series data by modeling the time component, improving reference gene selection for gene expression analysis.

LIMBR reduces batch effects and imputes missing data in time series proteomics by modeling experimental structure and circadian patterns.