Stefan Schuster

This study uses dynamic optimization to analyze host-pathogen interactions in invasive aspergillosis, identifying key roles for epithelial cells and neutrophils in immune response and fungal virulence.

The paper presents a mathematical model of molecular crypsis by pathogenic fungi using human factor H, finding that pathogen abundance relative to host cells determines the success of crypsis and potential autoreactivity.

ModuleDiscoverer identifies regulatory modules in protein-protein interaction networks using gene expression data and validates them with SNP data from GWAS.