Ina Maja Vorberg

This study shows HSV-1 fusion proteins enable cell-to-cell spread of pathological Tau, suggesting latent HSV-1 infection may contribute to neurodegenerative disease progression.

This study shows endogenous retroviruses enhance prion-like spreading of protein aggregates between cells, suggesting antiviral drugs could inhibit disease progression.

The paper shows viral ligand-receptor interactions enhance the spread of protein misfolding via extracellular vesicles and direct cell contact.