Author

Nirmala Prabu Gonsalves

Northwestern UniversityORCID 0000-0003-0722-6099Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:scRNA-seq· 2 Research Focus:cell-cell communication· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Adam L. Karami· 2 shared Deborah R. WinteriD· 2 shared Dustin A. CarlsoniD· 2 shared John Erik PandolfinoiD· 2 shared Kelly A. WhelaniD· 2 shared Margarette H. ClevengeriD· 2 shared Marie‐Pier TétreaultiD· 2 shared Cenfu WeiiD· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 43 mutual co-author links

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2 shared papersImmunology

Adam L. Karami

Mutual co-authors in cluster

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2 papers

Recent·Most cited

ImmunologyJournal of Allergy and Clinical ImmunologyPeer-reviewedJul 10, 2024

Esophageal epithelial Ikkβ deletion promotes eosinophilic esophagitis in experimental allergy mouse model

Margarette H. Clevenger, Cenfu Wei, Adam L. Karami…·Marie‐Pier Tétreault

This study shows that deleting Ikkβ in esophageal epithelial cells worsens eosinophilic esophagitis in a mouse model, highlighting the importance of IKKβ/NFκB signaling in preventing allergic inflammation.

4 citations

mousescRNA-seq

𝕏 PostBluesky

Molecular BiologybioRxivPreprintApr 20, 2023

Suprabasal cells retaining stem cell identity programs drive basal cell hyperplasia in eosinophilic esophagitis

Margarette H. Clevenger, Adam L. Karami, Dustin A. Carlson…·Marie‐Pier Tétreault

2 citations

humanscRNA-seq

𝕏 PostBluesky