Balázs Pósfai

The paper investigates how microglia change their phenotype in acute brain slices and how these changes affect neuronal network activity.

This study identifies microglia as key regulators of cerebral blood flow and neurovascular coupling through P2Y12R-mediated mechanisms in mice and humans.

This study identifies somatic purinergic junctions between microglia and developing neurons across developmental stages, suggesting they facilitate communication and regulation of neurogenesis.

This study shows that microglial NKCC1 regulates ion homeostasis, inflammation, and brain injury through cell-autonomous mechanisms.

The paper identifies specialized somatic microglia-neuron junctions that enable microglia to monitor and protect neuronal function through purinergic signaling.

The paper reveals that hippocampal cholinergic terminals form synapses and co-transmit GABA and acetylcholine via distinct vesicles, influencing hippocampal activity and potential therapeutic strategies.

The paper shows that mitochondrial ultrastructure in axons correlates with synaptic performance in mice and humans, indicating activity-dependent plasticity of mitochondria in response to synaptic demand.