Author

Karl‐Heinz Storbeck

Stellenbosch UniversityORCID 0000-0003-1669-6383Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Biochemistry· 1 Molecular Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Approach:experimental assay· 2 Assay:proteomics· 2 Research Focus:benchmarking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:metabolomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Eka MelsoniD· 2 shared Lina SchifferiD· 2 shared Wiebke ArltiD· 2 shared AE TayloriD· 1 shared Amarah V. AnthonyiD· 1 shared Anastasia Arvaniti· 1 shared Andrea WagenfeldiD· 1 shared Antonio M. A. MirandaiD· 1 shared

Activity by Year

2021-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersBiochemistry

Eka MelsonORCID

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Author profile →

3 papers

Recent·Most cited

BiochemistrybioRxivPreprintOct 29, 2026

Optimisation and Validation of a UHPLC-MS/MS Assay Using Timed Post-Column Ammonium Fluoride Infusion for Sensitive Serum Steroids Quantitation

Věra Dosedělová, James M. Hawley, Eka Melson…·Wiebke Arlt

The paper develops and validates a UHPLC-MS/MS assay for quantifying 18 steroids in serum, showing improved sensitivity and applied it to healthy individuals.

DOI 10.64898/2026.10.27.762247

humanmetabolomics

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PhysiologybioRxivPreprintMar 30, 2026

Inhibition of the androgen-activating enzyme AKR1C3 selectively decreases systemic and intra-adipose 11-oxygenated androgens in women

Lina Schiffer, Amarah V. Anthony, Laura B. L. Wittemans…·Wiebke Arlt

Inhibition of AKR1C3 reduces 11-oxygenated androgens but not classic androgens in women.

DOI 10.64898/2026.03.27.714735

humanproteomics

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Molecular BiologyJournal of Molecular EndocrinologyPeer-reviewedJan 27, 2021

Differential activity and expression of human 5β-reductase (AKR1D1) splice variants

Nathan Appanna, Hylton Gibson, Elena Gangitano…·Nikolaos Nikolaou

This study examines the differential expression and activity of three AKR1D1 splice variants in human tissues and cells, showing that AKR1D1-002 is the main functional variant involved in steroid hormone metabolism.

11 citations

humanproteomics

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