NeurosciencebioRxivPreprint
Improvement of Behavioral Deficits in a Mouse Model of Fragile X Syndrome by Restoring α7 nAChR Hypofunction Associated with Aberrant Ly6H Expression
Sarah Goebel, Dylann Cordova-Martinez, Vytas K. Verselis, Anna Francesconi
This study identifies 7 nAChR hypofunction as a therapeutic target in FXS by showing that restoring 7 nAChR function improves behavioral deficits in Fmr1KO mice.
DOI 10.1101/2024.11.01.621616