Author

Ben Kinnersley

Institute of Cancer ResearchORCID 0000-0003-1783-6296Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 3 Approach:statistical model· 3 Research Focus:variant calling· 2 Assay:whole-genome sequencing· 2 Research Focus:clustering· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Ian P.M. TomlinsoniD· 3 shared Richard S. HoulstoniD· 3 shared Andrea SottorivaiD· 2 shared Andreas J GruberiD· 2 shared Anna FrangouiD· 2 shared Boris NoyvertiD· 2 shared Clare TurnbulliD· 2 shared Daniel ChubbiD· 2 shared

Activity by Year

2020-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersGenetics

Ian P.M. TomlinsonORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

GenomicsbioRxivPreprintJun 19, 2026

Whole genome sequencing of endometrial cancer identifies novel subgroups, drivers, and actionable alterations

Sebastian Meyer, Ben Kinnersley, Katarzyna Kedzierska…·David N. Church

This study uses whole genome sequencing of endometrial cancers to identify novel subgroups, drivers, and actionable alterations, improving precision medicine approaches.

DOI 10.64898/2026.06.15.730391

humanwhole-genome sequencing

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GeneticsbioRxivPreprintApr 27, 2021

Germline loss-of-function variants in the base-excision repair gene MBD4 cause a Mendelian recessive syndrome of adenomatous colorectal polyposis and acute myeloid leukaemia

Claire Palles, Edward Chew, Judith E. Grolleman…·Richarda M. de Voer

3 citations

humanDNA methylation

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GeneticsNature CommunicationsPeer-reviewedJul 3, 2020

Assessment of Polygenic Architecture and Risk Prediction based on Common Variants Across Fourteen Cancers

Yan Zhang, Amber N. Hurson, Haoyu Zhang…·Colon Cancer Family Registry (CCFR)

This study assesses polygenicity and risk prediction using common variants across 14 cancers, finding varying numbers of susceptibility variants and risk score effectiveness.

137 citationstop 1%

humanGWAS summary statistics

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