Author

Gregory C. Rogers

University of ArizonaORCID 0000-0003-1870-1713Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Approach:statistical model· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:clustering· 1

Frequent Co-Authors

Anne E. CressiD· 2 shared John M. RyniaweciD· 2 shared Alejandro Recio‐BoilesiD· 1 shared Alex Patrick FarrelliD· 1 shared Amber N. ClementsiD· 1 shared Anastasia Amoiroglou· 1 shared Andrea L. CasillasiD· 1 shared Brian J. GallettaiD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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2 shared papersCancer Biology

Anne E. CressORCID

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3 papers

Recent·Most cited

Cell BiologybioRxivPreprintMay 5, 2026

A MOPD II-associated Pericentrin variant disrupts PACT domain dimerization and pericentriolar material recruitment

Makenzie Thomas, Brian J. Galletta, John M. Ryniawec…·Nasser M. Rusan

This study models a MOPD II-associated Pericentrin variant in Drosophila, showing it disrupts dimerization and pericentriolar material recruitment, leading to developmental and cellular defects.

DOI 10.64898/2026.05.01.722250

Drosophilaexperimental assay

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Cancer BiologyBritish Journal of CancerPeer-reviewedJan 7, 2026

Transient centrosome loss in cultured prostate epithelial cells induces chromosomal instability to produce an oncogenic genotype that correlates with poor clinical outcomes

Jiawen Yang, John M. Ryniawec, Diogo de Oliveira Pessôa…·Gregory C. Rogers

The paper shows that transient centrosome loss in prostate cells causes chromosomal instability, leading to oncogenic changes linked to poor clinical outcomes.

3 citationstop 3%

humanwhole-genome sequencing

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Cancer BiologyCancer Immunology ResearchPeer-reviewedFeb 21, 2025

Inhibition of PIM kinase in tumor associated macrophages suppresses inflammasome 1 activation and sensitizes prostate cancer to immunotherapy

Amber N. Clements, Andrea L. Casillas, Caitlyn E. Flores…·Noel A. Warfel

This study shows that inhibiting PIM kinases in tumor-associated macrophages reduces inflammasome activation and improves response to immunotherapy in prostate cancer.

13 citationstop 4%

mousebulk RNA-seq

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