Yun Liu

This study shows that loss of ACTA1 in mice delays the switch from γ-AChR to ε-AChR at the neuromuscular junction, affecting postsynaptic receptor maturation despite normal presynaptic function.

This paper shows that KIAA1109 is essential for mouse survival and proper neuromuscular junction development and function.

This study examines structural and functional changes in neuromuscular junctions of Acta1+/Ki mice, revealing impairments in synapse structure and transmission.