Author

Fabrizio Biundo

Albert Einstein College of MedicineORCID 0000-0003-2264-1872Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

Dennis W. DicksoniD· 2 shared E. Richard StanleyiD· 2 shared Gabriel G. L. Shlager· 2 shared Harmony C. Ketchum· 2 shared Michael A. DetureiD· 2 shared Violeta ChiţuiD· 2 shared Zbignew K. Wszolek· 2 shared Andrew L. CroxfordiD· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersNeuroscience

Dennis W. DicksonORCID

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintFeb 21, 2022

Granulocyte Colony Stimulating Factor causes cerebellar deficits and anxiety in a mouse model of CSF-1 receptor-related leukoencephalopathy

Fabrizio Biundo, Violeta Chiţu, Jaafar O. Tindi…·E. Richard Stanley

This study shows that reducing Csf3 in Csf1r+/- mice improves anxiety, motor coordination, and social interaction but not cognitive deficits, and reduces microglial activation in the cerebellum.

DOI 10.1101/2022.02.21.481325

mouseexperimental assay

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NeurosciencebioRxivDec 23, 2019

Microglial homeostasis requires balanced CSF-1/CSF-2 receptor signaling

Violeta Chiţu, Fabrizio Biundo, Gabriel G. L. Shlager…·E. Richard Stanley

This study shows that reducing CSF-2 in Csf1r+/- mice rescues behavioral and histopathological deficits in ALSP by preventing microgliosis and transcriptomic changes, suggesting CSF-2 as a therapeutic target.

DOI 10.1016/j.celrep.2020.02.028

mousehuman

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