Author

Joseph A. Giovinazzo

University of Colorado AnschutzORCID 0000-0003-2608-7143Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Biochemistry· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Assay:proteomics· 1 Assay:spatial transcriptomics· 1

Frequent Co-Authors

Rui ZhaoiD· 2 shared Shasha ShiiD· 2 shared Xueni LiiD· 2 shared Zhiling Kuang· 2 shared Anthony J. SaviolaiD· 1 shared Brian Kosmyna· 1 shared Christopher R. TrottaiD· 1 shared J. Matthew TaliaferroiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersBiochemistry

Rui ZhaoORCID

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2 papers

Recent·Most cited

BiochemistryNature CommunicationsPeer-reviewedJul 16, 2026

The U1 snRNP protein U1C and Helix H of U1 snRNA are critical for small molecule splicing modulator function

Zhiling Kuang, Brian Kosmyna, Xueni Li…·Rui Zhao

This study shows that U1C and Helix H of U1 snRNA are essential for branaplam's effect on weak 5 splice site recognition, while risdiplam requires additional factors for activity.

2 citations

spatial transcriptomicsexperimental assay

𝕏 PostBluesky

BiochemistryRNAPeer-reviewedApr 30, 2024

Selected humanization of yeast U1 snRNP leads to global suppression of pre-mRNA splicing and mitochondrial dysfunction in the budding yeast

Subbaiah Chalivendra, Shasha Shi, Xueni Li…·Rui Zhao

The paper humanizes yeast U1 snRNP components to study 5 ss recognition, finding that U1C ZnF substitution causes splicing defects and mitochondrial dysfunction.

6 citations

yeastproteomics

𝕏 PostBluesky