Rachel Templin

The paper investigates the life cycle and morphogenetic differentiation of decoupled cells in O. huxleyi, showing they can revert to calcified form without viral pressure and exhibit distinct transcriptomic and ultrastructural features.

This study identifies biallelic HMGCS1 variants as a novel cause of rigid spine syndrome, showing hypomorphic effects through functional and clinical analyses.

This study maps the central complex neuronal circuitry in bumblebees, identifying conserved and divergent features compared to fruit flies.