Tineke Grent-‘t-Jong

This study conducts a meta-analysis of GABAergic interneuron deficits in schizophrenia, finding reductions in parvalbumin and somatostatin interneurons, particularly in layers 3/4, and links these deficits to bottom-up processing impairments.

This study identifies large-scale neural dynamics changes in psychosis patients that resemble those from GABA-A and NMDA receptor manipulations and correlate with clinical symptoms.

This study investigates the role of GABAergic inhibition in the 40 Hz auditory steady-state response (ASSR) using MEG and pharmacological interventions, finding that GABAA receptor agonists increase ASSR amplitude while NMDA blockers have no effect.