André M. Bastos

This study uses human iEEG recordings to test predictive routing models during audiovisual speech violations, finding support for gamma and alpha-beta oscillation interactions in predictive processing.

This paper introduces GSDR, a genetic algorithm for training nonlinear neural models to reproduce oscillatory dynamics, showing it can achieve spectral and circuit-level phenotypes with reduced manual tuning.

This study uses control-theoretic methods to show that propofol anesthesia reduces interareal control in the brain, leading to a directional breakdown of cortical control.

The paper compares predictive coding, routing, and autoencoders using LFP data to determine their role in visual search task neural dynamics.

The paper shows that different anesthetics destabilize neural dynamics similarly, affecting recovery and low-frequency power, and linking this to unconsciousness.

The paper tests predictive processing hypotheses in mouse visual cortex using LFPs from multi-area laminar recordings during a visual oddball task, finding that gamma, beta, and theta bands signal different types of prediction errors.

This study conducts a meta-analysis of GABAergic interneuron deficits in schizophrenia, finding reductions in parvalbumin and somatostatin interneurons, particularly in layers 3/4, and links these deficits to bottom-up processing impairments.

The paper reaffirms the ubiquity of a spectrolaminar motif across multiple datasets and introduces an improved tool to identify it.

This study investigates how propofol-induced loss of consciousness affects neural oscillations and spiking activity in monkeys during auditory processing, finding disrupted alpha/beta inhibition and enhanced gamma/spiking.

This study challenges hierarchical predictive coding models by showing that prediction error responses in visual cortex do not involve feedforward processing and that predictive suppression is not mediated by inhibitory interneurons.

This study uses DeLASE to show that propofol anesthesia reduces dynamic stability in macaque cortex, mirroring destabilized linear systems.

The paper identifies a conserved cellular-to-spectral rule in primate cortex that predicts the spectrolaminar motif across brain areas and validates it in schizophrenia post-mortem data.

The paper identifies a consistent spectrolaminar pattern of LFP power across primate cortex, showing distinct frequency gradients in superficial and deep layers.

The paper models selective visual attention in macaques using Dynamic Causal Modeling to show that intrinsic V1 modulation explains attention effects in V1-V4 communication through changes in inhibition.

This study compares information content in local field potentials (LFP) and near-field LFP (nfLFP) to determine which better indexes local neural computations, finding nfLFP more informative for stimulus processing.

This paper introduces a physical neural mass model framework to infer oscillatory generator locations from laminar electrophysiological recordings in macaque monkeys.

The paper analyzes spike-field causal flow in different cortical layers and areas, finding frequency-specific laminar organization and directional information flow.

This study investigates how propofol induces unconsciousness in non-human primates and how thalamic stimulation reverses its effects by altering neural activity patterns.

The paper investigates how anatomical connection strength relates to rhythmic synchronization in macaque cortex, finding that strength predicts synchronization and vice versa, with frequency and direction specificity.

The paper investigates predictive coding in the brain by analyzing neural activity patterns in different cortical areas during a visual task, finding distinct frequency-specific routing mechanisms.

The paper examines oscillatory dynamics across different cortical areas in monkeys, finding that frequencies increase with cortical hierarchy while maintaining similar relationships to spiking activity.

This study identifies neural signatures of unconsciousness and shows that thalamic stimulation can reverse these features in anesthetized non-human primates.