Author

Eun‐Jeong Yang

Icahn School of Medicine at Mount SinaiORCID 0000-0003-3190-8604Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:benchmarking· 1 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

Giulio Maria PasinettiiD· 3 shared Kyle J. TrageseriD· 2 shared Molly S. EstilliD· 2 shared Umar Haris IqbaliD· 2 shared Brian MathewiD· 1 shared Chad SmithiD· 1 shared Elizabeth Griggs· 1 shared Francis J. HermaniD· 1 shared

Activity by Year

2022-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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3 shared papersNeuroscience

Giulio Maria PasinettiORCID

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3 papers

Recent·Most cited

NeuroscienceMolecular Nutrition & Food ResearchPeer-reviewedJul 13, 2023

The bioactive dietary polyphenol preparation alleviates depression and anxiety-like behaviors by modulating the regional heterogeneity of microglia morphology.

Eun‐Jeong Yang, Tal Frolinger, Susan Westfall…·Giulio Maria Pasinetti

This study shows that a grape-derived polyphenol preparation reduces depression and anxiety-like behaviors in mice by altering microglia morphology and modulating inflammatory pathways.

13 citations

mouseexperimental assay

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NeuroscienceeLifePeer-reviewedJun 28, 2023

Molecular and cellular similarities in the brain of SARS-CoV-2 and Alzheimer's disease individuals

Elizabeth Griggs, Kyle J. Trageser, Sean X. Naughton…·Giulio Maria Pasinetti

This study compares transcriptional and cellular changes in brain regions of SARS-CoV-2 infected, Alzheimer's disease, and SARS-CoV-2 infected Alzheimer's disease individuals to identify shared neuroinflammatory and blood-brain barrier alterations.

23 citations

humanbulk RNA-seq

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NeurosciencebioRxivPreprintJan 27, 2022

Inflammasome mediated neuronal-microglial crosstalk: a therapeutic substrate for the familial C9orf72 variant of the frontotemporal dementia/amyotrophic lateral sclerosis

Kyle J. Trageser, Chad Smith, Eun‐Jeong Yang…·Giulio Maria Pasinetti

This study investigates the role of the NLRP3 inflammasome in C9orf72 FTD/ALS pathogenesis, finding that its inhibition improves survival and reduces neurodegeneration.

DOI 10.1101/2022.01.27.478078

mouseexperimental assay

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