Author

Eric Jay Small

University of California, San FranciscoORCID 0000-0003-3191-6268Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Approach:statistical model· 1

Frequent Co-Authors

David Alan QuigleyiD· 2 shared Martin SjöströmiD· 2 shared Meng ZhangiD· 2 shared Raunak ShresthaiD· 2 shared Abhilash BarpandaiD· 1 shared Aidan Winters· 1 shared Aishwarya SubramanianiD· 1 shared Arun P. WiitaiD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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2 shared papersCancer Biology

David Alan QuigleyORCID

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2 papers

Recent·Most cited

Cancer BiologyNature GeneticsPeer-reviewedNov 5, 2025

Genome-wide CRISPR screens identify PTGES3 as a novel AR modulator

Haolong Li, James E. Melnyk, Becky Xu Hua Fu…·Felix Yi-Chung Feng

This study uses CRISPR screens to identify PTGES3 as a novel modulator of AR protein levels and function in prostate cancer, showing its role in therapy resistance.

10 citationstop 2%

humangenetic perturbation / CRISPR

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Cancer BiologybioRxivPreprintJun 2, 2024

A prostate cancer gastrointestinal transcriptional phenotype may be associated with diminished response to AR-targeted therapy

Aishwarya Subramanian, Meng Zhang, Marina Nasrin Sharifi…·Martin Sjöström

This study identifies a gastrointestinal gene expression phenotype in metastatic castration-resistant prostate cancer that correlates with reduced response to androgen receptor inhibitors.

DOI 10.1101/2024.06.02.595931

humanbulk RNA-seq

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