Henrik Sahlin Pettersen

This study identifies divergent brain pathology in preterm infants with NEC, showing distinct cellular and molecular changes in the pons and cerebellum.

The paper shows that prematurity disrupts cerebellar development by altering developmental trajectories and causing lineage-specific asynchrony in cell maturation.

The paper investigates cerebellar dysfunction in SIDS by integrating multi-omic data, finding metabolic and developmental abnormalities linked to neuroinflammation and impaired autonomic responses.

The paper shows that slow outer hair cell contractions enable fast electrical responses to intense sound, revealing a kinetic relationship essential for cochlear protection and temporal fidelity.