Author

Samuel Liégeois

Centre National de la Recherche ScientifiqueORCID 0000-0003-3228-7678Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Immunology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:Drosophila· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:C. elegans· 1

Frequent Co-Authors

Dominique FerrandoniD· 2 shared Abdul Hakkim· 1 shared Adrien FranchetiD· 1 shared Bo ZhangiD· 1 shared Eliana Drenkard· 1 shared Frederick M. Ausubel· 1 shared Jianqiong HuangiD· 1 shared Jianwen Yang· 1 shared

Activity by Year

2017-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersImmunology

Dominique FerrandonORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

ImmunologyEMBO ReportsPeer-reviewedJan 9, 2026

Distinct Bomanins at the Drosophila 55C locus function in resistance and resilience to infections

Yanyan Lou, Bo Zhang, Zhiyuan Zhang…·Dominique Ferrandon

The paper investigates the roles of distinct Bomanin peptides from the 55C locus in Drosophila resistance and resilience to infections, finding overlapping and specific functions against different pathogens.

8 citationstop 1%

Drosophilaexperimental assay

𝕏 PostBluesky

ImmunologybioRxivPreprintJul 26, 2017

RhlR, but not RhlI, allows P. aeruginosa bacteria to evade Drosophila Tep4- mediated opsonization

Samantha Haller, Adrien Franchet, Abdul Hakkim…·Dominique Ferrandon

This study shows that RhlR, not RhlI, enables P. aeruginosa to evade Drosophila Tep4-mediated opsonization, highlighting a RhlI-independent immune evasion mechanism.

DOI 10.1101/165530

DrosophilaC. elegans

𝕏 PostBluesky