Author

Rebecca C. Wyatt

University of ExeterORCID 0000-0003-3345-3383Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetic & Genomic Medicine· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:imaging· 1 Approach:statistical model· 1

Frequent Co-Authors

Christine S. FlaxmaniD· 2 shared Noel G. MorganiD· 2 shared Sarah J. RichardsoniD· 2 shared Ana Tangari· 1 shared Andrew Tym HattersleyiD· 1 shared Antonia DastamaniiD· 1 shared Birgit M. Rami-MerhariD· 1 shared Catarina LimbertiD· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersPathology

Christine S. FlaxmanORCID

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2 papers

Recent·Most cited

PathologybioRxivPreprintApr 11, 2025

✦ In-depth digest

Small things matter: Lack of extra-islet beta cells in Type 1 diabetes

Kathryn Murrall, Teifion Luckett, Christiana Lekka…·Sarah J. Richardson

Image analysis of 262,832 pancreatic endocrine objects across 220 donors reveals that small beta-cell-only clusters are abundant in early childhood but virtually absent in type 1 diabetes.

11 citations

humanimaging

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Genetic & Genomic MedicineNature GeneticsPeer-reviewedNov 1, 2022

A novel disease mechanism leading to the expression of a disallowed gene in the pancreatic beta-cell identified by non-coding, regulatory mutations controlling HK1

Matthew Neil Wakeling, Nick D. L. Owens, Jessica R. Hopkinson…·Sarah Elizabeth Flanagan

The paper identifies non-coding mutations in a regulatory element of HK1 that lead to inappropriate expression in pancreatic beta-cells and cause congenital hyperinsulinism.

66 citationstop 1%

humanbulk RNA-seq

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