Author

Michael G. Ison

Northwestern UniversityORCID 0000-0003-3347-9671Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetic & Genomic Medicine· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Approach:statistical model· 2 Approach:deep learning· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Assay:proteomics· 1

Frequent Co-Authors

Abdul Rouf BandayiD· 1 shared Adam J. GehringiD· 1 shared Alicia K. SmithiD· 1 shared Amy A HutchinsoniD· 1 shared Andrea N. Burnett‐HartmaniD· 1 shared Andrew Daniel HoffmanniD· 1 shared Anurag VermaiD· 1 shared Benedito Antônio Lopes da FonsecaiD· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperGenetic & Genomic Medicine

Abdul Rouf BandayORCID

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2 papers

Recent·Most cited

ImmunologyiSciencePeer-reviewedSep 17, 2025

Novel antibody language model accelerates IgG screening and design for broad-spectrum antiviral therapy

Hannah Faisal Almubarak, Wuwei Tan, Andrew Daniel Hoffmann…·Yang Shen

This paper introduces an antibody language model (AbLM) to accelerate IgG screening and design for broad-spectrum antiviral therapy, achieving higher precision in predicting variant-resistant antibodies.

1 citation

humanproteomics

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Genetic & Genomic MedicineNature GeneticsPeer-reviewedJul 14, 2022

Genetic regulation of OAS1 nonsense-mediated decay underlies association with risk of severe COVID-19

Abdul Rouf Banday, Megan Lynn Stanifer, Oscar Flórez-Vargas…·Ludmila Prokunina‐Olsson

The paper identifies a Neandertal introgressed haplotype in the OAS1 gene associated with increased risk of severe COVID-19 and shows it impairs SARS-CoV-2 clearance through genetic regulation of OAS1 expression.

130 citationstop 1%

humanwhole-genome sequencing

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