Author

Valeria Bica

University of Rome Tor VergataORCID 0000-0003-3366-5562Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2 Systems Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:proteomics· 3 Research Focus:differential expression· 2 Research Focus:drug response / drug discovery· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Approach:experimental assay· 1

Frequent Co-Authors

Dimitrios MougiakakosiD· 3 shared Francesca SaccoiD· 3 shared Giorgia MassacciiD· 3 shared Livia PerfettoiD· 3 shared Thomas FischeriD· 3 shared Martin BöttcheriD· 2 shared Patrizia ChiusoloiD· 2 shared Veronica VenafraiD· 2 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

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3 shared papersCancer Biology

Dimitrios MougiakakosORCID

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3 papers

Recent·Most cited

Cancer BiologyLeukemiaPeer-reviewedJun 9, 2026

Organelle proteomics reveals novel metabolic vulnerabilities in FLT3-ITD cells

Valeria Bica, Anna Francesca Pacile, Martin Boettcher…·Francesca Sacco

The paper uses proteomics to identify metabolic vulnerabilities in FLT3-ITD AML cells, showing lipid restriction restores midostaurin sensitivity.

DOI 10.1038/s41375-026-03000-6

humanproteomics

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Cancer BiologybioRxivPreprintOct 7, 2025

Targeting Kinases to Reshape the Immunopeptidome landscape in Chronic Myeloid Leukemia

Veronica Venafra, Maria Wahle, Giorgia Massacci…·Francesca Sacco

This study investigates how kinase inhibitors reshape the immunopeptidome in CML by altering HLA-I peptide presentation through three molecular mechanisms.

DOI 10.1101/2025.10.07.680859

humanproteomics

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Systems BiologybioRxivPreprintJan 8, 2025

Phosphoproteomics reveals novel BCR::ABL1-independent mechanisms of resistance in chronic myeloid leukemia

Valeria Bica, Veronica Venafra, Giorgia Massacci…·Francesca Sacco

The paper identifies BCR::ABL1-independent survival mechanisms in CML using phosphoproteomics and computational analysis, highlighting FLT3 as a therapeutic target for TKI-resistant cases.

1 citation

humanproteomics

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